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DNA-binding-deficient Hand2 dimerizes with Tcf3a to control zebrafish cardiogenesis

The bHLH transcription factor Hand2 positively regulates pdgfra expression to promote early cardiogenesis in zebrafish. Hand2 can function in this process independently of direct DNA binding by interacting with Tcf3a, another bHLH transcription factor.

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Fig. 1: Hand2 regulates pdgfra expression to mediate early cardiogenesis.

References

Srivastava, D. et al. Regulation of cardiac mesodermal and neural crest development by the bHLH transcription factor, dHAND. Nat. Genet. 16, 154–160 (1997). This paper first reported the morphological defects in dHAND (Hand2)-mutant mouse embryos.

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Liu, N. et al. DNA binding-dependent and -independent functions of the Hand2 transcription factor during mouse embryogenesis. Development 136, 933–942 (2009). This paper showed that the DNA-binding domain of Hand2 is not required for early mouse cardiogenesis.

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de Soysa, T. Y. et al. Single-cell analysis of cardiogenesis reveals basis for organ-level developmental defects. Nature 572, 120–124 (2019). This paper provided the embryonic mouse Hand2-mutant heart single-cell RNA-sequencing datasets used in our paper.

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This is a summary of: Xu, Y. et al. PDGFRA is a conserved HAND2 effector during early cardiac development. Nat. Cardiovasc. Res. https://doi.org/10.1038/s44161-024-00574-1 (2024).

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DNA-binding-deficient Hand2 dimerizes with Tcf3a to control zebrafish cardiogenesis. Nat Cardiovasc Res (2024). https://doi.org/10.1038/s44161-024-00589-8

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Published:10 December 2024

DOI:https://doi.org/10.1038/s44161-024-00589-8

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