Correction to: Nature https://doi.org/10.1038/s41586-022-05074-5 Published online 17 August 2022
In the version of the article initially published, Fig. 3j and Extended Data Fig. 5a contained errors that were generated during figure assembly. In both cases, the initially published accompanying source data file contains the correct data. None of the paper’s interpretations are affected by this correction.
Figure 3j contained an error in the lrpl-1(lof) lrp2(lof) mutant data, which were mistakenly graphed and inadvertently duplicated from the overexpressed DMA-1 condition of Fig. 1p. This error only pertains to the lrpl-1(lof) lrp2(lof) mutant data. Data for wild type and degt-1(lof) were intentionally re-graphed between Fig. 1p and Fig. 3j as specified in the published Fig. 3 legend. The corrected graph is shown below in Fig. 1, together with the incorrect published graph, for transparency to the readers. This error did not affect any conclusions because the source data were correct, and the mistaken bar graph in question was from a mutant phenotype with a very similar defect (76.67% vs 78.33% of animals responding).
Fig. 1: Original and corrected Fig. 3j
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Extended Data Fig. 5a contained an incorrect display micrograph for the DMA-1(ΔYFGI) mutant. This error occurred during figure assembly, and the corresponding quantification and raw data for this experiment, which are graphed in Extended Data Fig. 5b, are correct. The corrected figure panel is shown below in Fig. 2, together with the incorrect published display panel, for transparency to the readers. This error did not affect any conclusions because the source data were correct, and the mistaken display panel was from a statistically indistinguishable genotype.
Fig. 2: Original and corrected Extended Data Fig. 5a
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Authors and Affiliations
Howard Hughes Medical Institute, Department of Biology, Stanford University, Stanford, CA, USA
Kelsie Eichel, Caitlin A. Taylor & Kang Shen
Department of Pathology, Stanford University School of Medicine, Stanford, CA, USA
Takeshi Uenaka & Marius Wernig
Institute for Stem Cell Biology and Regenerative Medicine, Stanford University School of Medicine, Stanford, CA, USA
Takeshi Uenaka & Marius Wernig
University of Bordeaux, CNRS, Interdisciplinary Institute for Neuroscience, Bordeaux, France
Vivek Belapurkar & David Perrais
Department of Molecular and Cellular Physiology, Stanford University, Stanford, CA, USA
Rui Lu & Thomas C. Südhof
Howard Hughes Medical Institute, Stanford University School of Medicine, Stanford, CA, USA
Rui Lu & Thomas C. Südhof
Nancy Pritzker Laboratory, Department of Psychiatry and Behavioral Sciences, Stanford University, Stanford, CA, USA
Rui Lu & Robert Malenka
Department of Biochemistry and Molecular Biology, University of Chicago, Chicago, IL, USA
Shouqiang Cheng, Joseph S. Pak & Engin Özkan
Grossman Institute of Neuroscience, Quantitative Biology and Human Behavior, University of Chicago, Chicago, IL, USA
Shouqiang Cheng, Joseph S. Pak & Engin Özkan
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Kelsie Eichel
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2. Takeshi Uenaka
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3. Vivek Belapurkar
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7. Caitlin A. Taylor
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8. Thomas C. Südhof
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9. Robert Malenka
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10. Marius Wernig
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11. Engin Özkan
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12. David Perrais
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13. Kang Shen
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Eichel, K., Uenaka, T., Belapurkar, V. et al. Author Correction: Endocytosis in the axon initial segment maintains neuronal polarity. Nature (2025). https://doi.org/10.1038/s41586-025-08837-y
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Published:18 March 2025
DOI:https://doi.org/10.1038/s41586-025-08837-y
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